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This paper introduces an approach to automatic generation of visually faultless facial morphs along with a proposal on how such morphs can be automatically detected. It is endeavored that the created morphs cannot be recognized as such with the naked eye and a reference automatic face recognition (AFR) system produces high similarity scores while matching a morph against faces of persons who participated in morphing. Automatic generation of morphs allows for creating abundant experimental data, which is essential (i) for evaluating the performance of AFR systems to reject morphs and (ii) for training forensic systems to detect morphs. Our first experiment shows that human performance to distinguish between morphed and genuine face images is close to random guessing. In our second experiment, the reference AFR system has verified 11.78% of morphs against any of genuine images at the decision threshold of 1% false acceptance rate. These results indicate that facial morphing is a serious threat to access control systems aided by AFR and establish the need for morph detection approaches. Our third experiment shows that the distribution of Benford features extracted from quantized DCT coefficients of JPEG-compressed morphs is substantially different from that of genuine images enabling the automatic detection of morphs.
The paper presents an observation and evaluation of the recovery process. Seven weeks after the fracture of the fibula and six weeks after surgery the measurement of the gait of the subject has been started. Inertial measurement units applied above both ankles are used to acquire kinematical data and three surface EMG at each leg to get information about the activity of three muscles. The intensity of the weekly tests has been increased in dependence on the healing progress. Both scenarios-walking on the floor and on the treadmill-were included in the experiments to obtain comparable data sets. Beside of the investigation of the healing process the paper is focused on symmetry/asymmetry properties of the gait. The results indicate that asymmetry is not only an essential property of pathologic gait but it is present in normal gait too, especially if short distances are evaluated. They may be caused by the influence of a dominant/injured leg, of the first/last steps, the natural variance of the gait or the daily fitness of the subject. Asymmetry is obviously in the contribution of both legs to a stride (step length, strike and lift angle, stand and swing phase).